Telogen effluvium is a diffuse hair shedding condition associated with disruptions in the normal hair growth cycle, particularly a premature shift of hair follicles from the active anagen growth phase into the resting telogen phase. Increased shedding commonly develops several weeks to months after periods of physiological stress, emotional stress, inflammatory burden, rapid dietary changes, nutrient insufficiency, illness recovery, sleep disruption, endocrine stress, or metabolic imbalance. Hair follicles are highly metabolically active structures that require continuous cellular energy production, amino acid availability, antioxidant protection, micronutrient support, oxygen delivery, and balanced endocrine signaling to maintain normal growth activity. Stress-related signaling pathways may influence follicular cycling through elevated cortisol exposure, inflammatory cytokine activity, oxidative stress accumulation, mitochondrial strain, and altered nutrient partitioning. Chronic activation of the hypothalamic-pituitary-adrenal axis may contribute to impaired follicular growth signaling, collagen support disruption, microvascular circulation changes, and increased oxidative burden surrounding the follicular environment. Hair follicles rely heavily on rapidly dividing keratinocyte populations that require adequate protein synthesis, mitochondrial ATP production, iron transport, zinc-dependent enzyme activity, and antioxidant defense systems. Oxidative stress and inflammatory signaling may further contribute to follicular stress responses. Reactive oxygen species can impair cellular membranes, mitochondrial efficiency, and structural proteins associated with normal hair shaft formation. Reduced antioxidant defense activity involving glutathione systems, superoxide dismutase, catalase, and selenium-dependent enzymes may increase susceptibility to cellular stress within scalp tissues. Nutritional insufficiency involving iron, zinc, selenium, vitamin C, folate-related pathways, amino acids, and antioxidant-rich whole foods may also influence follicular resilience and keratin production. A whole food plant-based dietary pattern emphasizing legumes, leafy greens, colorful vegetables, berries, seeds, whole grains, mushrooms, and antioxidant-rich plant foods may help support normal follicular biology, mitochondrial activity, oxidative balance, microcirculation, and connective tissue integrity. Fiber-rich whole foods may support metabolic stability, inflammatory regulation, gut microbiome activity, and endocrine balance associated with healthy hair follicle cycling. Foods such as lentils, pumpkin seeds, spinach, kale, broccoli, strawberries, blueberries, quinoa, chickpeas, oats, walnuts, flax seeds, and green tea provide iron, zinc, selenium, vitamin C compounds, amino acids, polyphenols, lignans, catechins, carotenoids, and antioxidant phytochemicals associated with cellular protection and normal tissue maintenance. Sulfur-containing vegetables, polyphenol-rich berries, cruciferous vegetables, and flavonoid-containing whole foods may also support antioxidant response systems and scalp tissue integrity associated with normal hair growth biology.